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human gfap  (R&D Systems)


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    Structured Review

    R&D Systems human gfap
    Human Gfap, supplied by R&D Systems, used in various techniques. Bioz Stars score: 93/100, based on 18 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/human+gfap+duoset+elisa/Human+GFAP+DuoSet+ELISA/10__1016_slash_j__cej__2025__161118-297-7-9
    Average 93 stars, based on 18 article reviews
    human gfap - by Bioz Stars, 2026-11
    93/100 stars

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    Related Articles

    Enzyme-linked Immunosorbent Assay:

    Article Title: Clinical characteristics of double negative atypical inflammatory demyelinating disease: A prospective study.
    Article Snippet: .. Serum GFAP of double negative atypical IDD patients was quantified using commercial Human GFAP DuoSet ELISA (R&D Systems Minneapolis, MN, USA) by an independent investigator who was blinded to the result of TBA. ..

    Article Title: Altered metabolic function induced by amyloid-beta oligomers and PSEN1-mutations in iPSC-derived astrocytes
    Article Snippet: IL-6 and IL-8 levels in conditioned media were quantified using Quantikine immunoassays (HS600C and D8000C, R&D systems). .. GFAP was measured in CM using Human GFAP DuoSet ELISA (R&Dsystems, DY2594-05) as per manufacturer’s instructions. .. To quantify intracellular accumulation of IL-6 and IL-8, astrocytes (1×10 6 ) were incubated with protein transport inhibitor containing Brefeldin A (555029 Golgiplug, BD Biosciences) and Monensin (554724 Golgistop, BD Biosciences) following 48hrs treatment with Aβ oligomers or 4hrs with IL1β (10ng/mL, 200-01B Peprotech).

    Article Title: Serum Levels of S100B Protein and Myelin Basic Protein as a Potential Biomarkers of Recurrent Depressive Disorders.
    Article Snippet: .. Concentrations of calcium-binding protein (S100B), MBP and GFAP were determined in the blood serum by enzyme-linked immunosorbent assay using the DY1820-05 Human S100B DuoSet ELISA, DY4228-05 Human MBP DuoSet ELISA and DY2594-05 Human GFAP DuoSet ELISA manufactured by R&D Systems (Minneapolis, MN, USA). ..

    Article Title: Neuroimaging, wearable sensors, and blood-based biomarkers reveal hyperacute changes in the brain after sub-concussive impacts.
    Article Snippet: .. Blood-based biomarkers of brain injury included serum neurofilament light (NfL) concentrations (pg/mL) a biomarker of axonal injury and plasma glialfibrillary acidic protein (GFAP), an astrocyte injury biomarker, and were analyzed by Quanterix Corp using the NF-Light Simoa Assay Advantage Kit (Quanterix Corp, MA, USA) and R&D Systems human GFAP duoset ELISA (Bio-Techne, R&D Systems kit, Minneapolis, MN, USA) respectively. .. Plasma IL-6 (pg/mL) concentrations were determined using the Human IL6 Quantikine ELISA (Bio-Techne, R&D Systems high-sensitivity kit, Minneapolis, MN, USA).

    Article Title: Neuroimaging, wearable sensors, and blood-based biomarkers reveal hyperacute changes in the brain after sub-concussive impacts
    Article Snippet: .. Blood-based biomarkers of brain injury included serum neurofilament light (NfL) concentrations (pg/mL) a biomarker of axonal injury and plasma glial-fibrillary acidic protein (GFAP), an astrocyte injury biomarker, and were analyzed by Quanterix Corp using the NF-Light Simoa Assay Advantage Kit (Quanterix Corp, MA, USA) and R&D Systems human GFAP duoset ELISA (Bio-Techne, R&D Systems kit, Minneapolis, MN, USA) respectively. .. Blood-based biomarkers of brain injury included serum neurofilament light (NfL) concentrations (pg/mL) a biomarker of axonal injury and plasma glial-fibrillary acidic protein (GFAP), an astrocyte injury biomarker, and were analyzed by Quanterix Corp using the NF-Light Simoa Assay Advantage Kit (Quanterix Corp, MA, USA) and R&D Systems human GFAP duoset ELISA (Bio-Techne, R&D Systems kit, Minneapolis, MN, USA) respectively.

    Article Title: Clinical characteristics of double negative atypical inflammatory demyelinating disease: A prospective study
    Article Snippet: .. Serum GFAP of double negative atypical IDD patients was quantified using commercial Human GFAP DuoSet ELISA (R&D Systems Minneapolis, MN, USA) by an independent investigator who was blinded to the result of TBA. ..

    Article Title: Postmortem biochemistry of GFAP, NSE and S100B in cerebrospinal fluid and in vitreous humor for estimation of postmortem interval: a pilot study.
    Article Snippet: Postmortem interval (PMI) estimation is a major challenge in forensic pathology [1, 2].. The forensic pathologist is often called upon to gather all the information during crime scene investigations, deferring the involvement of forensic sciences experts to later stages.. The “Site inspection form” proposed by the Parma Institute of Legal Medicine, recently shared with the European Council of Legal Medicine, represents an attempt to overcome these logistical problems [3].

    Article Title: Serum Levels of S100B Protein and Myelin Basic Protein as a Potential Biomarkers of Recurrent Depressive Disorders
    Article Snippet: .. Concentrations of calcium-binding protein (S100B), MBP and GFAP were determined in the blood serum by enzyme-linked immunosorbent assay using the DY1820-05 Human S100B DuoSet ELISA, DY4228-05 Human MBP DuoSet ELISA and DY2594-05 Human GFAP DuoSet ELISA manufactured by R&D Systems (Minneapolis, MN, USA). ..

    Biomarker Discovery:

    Article Title: Neuroimaging, wearable sensors, and blood-based biomarkers reveal hyperacute changes in the brain after sub-concussive impacts.
    Article Snippet: .. Blood-based biomarkers of brain injury included serum neurofilament light (NfL) concentrations (pg/mL) a biomarker of axonal injury and plasma glialfibrillary acidic protein (GFAP), an astrocyte injury biomarker, and were analyzed by Quanterix Corp using the NF-Light Simoa Assay Advantage Kit (Quanterix Corp, MA, USA) and R&D Systems human GFAP duoset ELISA (Bio-Techne, R&D Systems kit, Minneapolis, MN, USA) respectively. .. Plasma IL-6 (pg/mL) concentrations were determined using the Human IL6 Quantikine ELISA (Bio-Techne, R&D Systems high-sensitivity kit, Minneapolis, MN, USA).

    Article Title: Neuroimaging, wearable sensors, and blood-based biomarkers reveal hyperacute changes in the brain after sub-concussive impacts
    Article Snippet: .. Blood-based biomarkers of brain injury included serum neurofilament light (NfL) concentrations (pg/mL) a biomarker of axonal injury and plasma glial-fibrillary acidic protein (GFAP), an astrocyte injury biomarker, and were analyzed by Quanterix Corp using the NF-Light Simoa Assay Advantage Kit (Quanterix Corp, MA, USA) and R&D Systems human GFAP duoset ELISA (Bio-Techne, R&D Systems kit, Minneapolis, MN, USA) respectively. .. Blood-based biomarkers of brain injury included serum neurofilament light (NfL) concentrations (pg/mL) a biomarker of axonal injury and plasma glial-fibrillary acidic protein (GFAP), an astrocyte injury biomarker, and were analyzed by Quanterix Corp using the NF-Light Simoa Assay Advantage Kit (Quanterix Corp, MA, USA) and R&D Systems human GFAP duoset ELISA (Bio-Techne, R&D Systems kit, Minneapolis, MN, USA) respectively.

    Article Title: Postmortem biochemistry of GFAP, NSE and S100B in cerebrospinal fluid and in vitreous humor for estimation of postmortem interval: a pilot study.
    Article Snippet: Postmortem interval (PMI) estimation is a major challenge in forensic pathology [1, 2].. The forensic pathologist is often called upon to gather all the information during crime scene investigations, deferring the involvement of forensic sciences experts to later stages.. The “Site inspection form” proposed by the Parma Institute of Legal Medicine, recently shared with the European Council of Legal Medicine, represents an attempt to overcome these logistical problems [3].

    Clinical Proteomics:

    Article Title: Neuroimaging, wearable sensors, and blood-based biomarkers reveal hyperacute changes in the brain after sub-concussive impacts.
    Article Snippet: .. Blood-based biomarkers of brain injury included serum neurofilament light (NfL) concentrations (pg/mL) a biomarker of axonal injury and plasma glialfibrillary acidic protein (GFAP), an astrocyte injury biomarker, and were analyzed by Quanterix Corp using the NF-Light Simoa Assay Advantage Kit (Quanterix Corp, MA, USA) and R&D Systems human GFAP duoset ELISA (Bio-Techne, R&D Systems kit, Minneapolis, MN, USA) respectively. .. Plasma IL-6 (pg/mL) concentrations were determined using the Human IL6 Quantikine ELISA (Bio-Techne, R&D Systems high-sensitivity kit, Minneapolis, MN, USA).

    Article Title: Neuroimaging, wearable sensors, and blood-based biomarkers reveal hyperacute changes in the brain after sub-concussive impacts
    Article Snippet: .. Blood-based biomarkers of brain injury included serum neurofilament light (NfL) concentrations (pg/mL) a biomarker of axonal injury and plasma glial-fibrillary acidic protein (GFAP), an astrocyte injury biomarker, and were analyzed by Quanterix Corp using the NF-Light Simoa Assay Advantage Kit (Quanterix Corp, MA, USA) and R&D Systems human GFAP duoset ELISA (Bio-Techne, R&D Systems kit, Minneapolis, MN, USA) respectively. .. Blood-based biomarkers of brain injury included serum neurofilament light (NfL) concentrations (pg/mL) a biomarker of axonal injury and plasma glial-fibrillary acidic protein (GFAP), an astrocyte injury biomarker, and were analyzed by Quanterix Corp using the NF-Light Simoa Assay Advantage Kit (Quanterix Corp, MA, USA) and R&D Systems human GFAP duoset ELISA (Bio-Techne, R&D Systems kit, Minneapolis, MN, USA) respectively.



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    Inclusion procedure of double negative atypical IDD, MS, AQP4‐IgG + NMOSD, and MOGAD. Of the 298 patients initially enrolled, 98 double‐negative atypical IDD, 73 AQP4‐IgG‐positive NMOSD, 23 MOGAD, and 63 MS patients were finally included in the analysis after rigorous screening. AQP4, aquaporin‐4; <t>GFAP,</t> glial fibrillary acidic protein; IDD, inflammatory demyelinating diseases; MOG, myelin oligodendrocyte glycoprotein; MOGAD, myelin oligodendrocyte glycoprotein antibody associated disease; MS, multiple sclerosis; NMOSD, neuromyelitis optica spectrum disorders.
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    Image Search Results


    Inclusion procedure of double negative atypical IDD, MS, AQP4‐IgG + NMOSD, and MOGAD. Of the 298 patients initially enrolled, 98 double‐negative atypical IDD, 73 AQP4‐IgG‐positive NMOSD, 23 MOGAD, and 63 MS patients were finally included in the analysis after rigorous screening. AQP4, aquaporin‐4; GFAP, glial fibrillary acidic protein; IDD, inflammatory demyelinating diseases; MOG, myelin oligodendrocyte glycoprotein; MOGAD, myelin oligodendrocyte glycoprotein antibody associated disease; MS, multiple sclerosis; NMOSD, neuromyelitis optica spectrum disorders.

    Journal: Annals of Clinical and Translational Neurology

    Article Title: Clinical characteristics of double negative atypical inflammatory demyelinating disease: A prospective study

    doi: 10.1002/acn3.52191

    Figure Lengend Snippet: Inclusion procedure of double negative atypical IDD, MS, AQP4‐IgG + NMOSD, and MOGAD. Of the 298 patients initially enrolled, 98 double‐negative atypical IDD, 73 AQP4‐IgG‐positive NMOSD, 23 MOGAD, and 63 MS patients were finally included in the analysis after rigorous screening. AQP4, aquaporin‐4; GFAP, glial fibrillary acidic protein; IDD, inflammatory demyelinating diseases; MOG, myelin oligodendrocyte glycoprotein; MOGAD, myelin oligodendrocyte glycoprotein antibody associated disease; MS, multiple sclerosis; NMOSD, neuromyelitis optica spectrum disorders.

    Article Snippet: Serum GFAP of double negative atypical IDD patients was quantified using commercial Human GFAP DuoSet ELISA (R&D Systems Minneapolis, MN, USA) by an independent investigator who was blinded to the result of TBA.

    Techniques:

    The specificity and sensitivity of the TBA using a rat brain indirect immunofluorescence. (A) The TBA results of the specimens that positive for AQP4‐IgG or GFAP‐IgG determined by CBA. (B) The TBA results of the specimens tested negative for AQP4‐IgG, GFAP‐IgG, and MOG‐IgG determined by CBA. AQP4, aquaporin‐4; CNS, central nervous system; IDD, inflammatory demyelinating diseases; MOGAD, myelin oligodendrocyte glycoprotein antibody‐associated disease; MS, multiple sclerosis; NIE, noninflammatory encephalopathy; NIM, noninflammatory myelopathy; NIPN, noninflammatory peripheral neuropathy; NMOSD, neuromyelitis optica spectrum disorders.

    Journal: Annals of Clinical and Translational Neurology

    Article Title: Clinical characteristics of double negative atypical inflammatory demyelinating disease: A prospective study

    doi: 10.1002/acn3.52191

    Figure Lengend Snippet: The specificity and sensitivity of the TBA using a rat brain indirect immunofluorescence. (A) The TBA results of the specimens that positive for AQP4‐IgG or GFAP‐IgG determined by CBA. (B) The TBA results of the specimens tested negative for AQP4‐IgG, GFAP‐IgG, and MOG‐IgG determined by CBA. AQP4, aquaporin‐4; CNS, central nervous system; IDD, inflammatory demyelinating diseases; MOGAD, myelin oligodendrocyte glycoprotein antibody‐associated disease; MS, multiple sclerosis; NIE, noninflammatory encephalopathy; NIM, noninflammatory myelopathy; NIPN, noninflammatory peripheral neuropathy; NMOSD, neuromyelitis optica spectrum disorders.

    Article Snippet: Serum GFAP of double negative atypical IDD patients was quantified using commercial Human GFAP DuoSet ELISA (R&D Systems Minneapolis, MN, USA) by an independent investigator who was blinded to the result of TBA.

    Techniques: Immunofluorescence

    TBA immunofluorescence patterns of autoantibodies against astrocytes, neurons, or oligodendrocyte in double negative atypical IDD patients. (A) An immunofluorescence pattern of antibodies from patients with double negative atypical IDD binding to astrocytes at rat cerebral cortex (green), similar to commercial AQP4‐IgG (red). (B) An immunofluorescence pattern of antibodies from patients with double negative atypical IDD binding to astrocytes at rat cerebral cortex (green), similar to commercial GFAP‐IgG (red). (C) An immunofluorescence pattern of antibodies from patients with double negative atypical IDD binding to neurons at rat cerebral cortex (green), similar to commercial NeuN‐IgG (red). (D) An immunofluorescence pattern of antibodies from patients with double negative atypical IDD binding to oligodendrocytes at rat cerebellum (green), similar to commercial MOG‐IgG (red). (E) Seronegative immunofluorescence pattern. Nuclei were counterstained with 4′,6‐diamidino‐2‐phenylindole (DAPI) (blue). Scale bars: 100 μm. AQP4, aquaporin‐4; GFAP, glial fibrillary acidic protein; MOG, myelin oligodendrocyte glycoprotein; NeuN, neuronal nucleus.

    Journal: Annals of Clinical and Translational Neurology

    Article Title: Clinical characteristics of double negative atypical inflammatory demyelinating disease: A prospective study

    doi: 10.1002/acn3.52191

    Figure Lengend Snippet: TBA immunofluorescence patterns of autoantibodies against astrocytes, neurons, or oligodendrocyte in double negative atypical IDD patients. (A) An immunofluorescence pattern of antibodies from patients with double negative atypical IDD binding to astrocytes at rat cerebral cortex (green), similar to commercial AQP4‐IgG (red). (B) An immunofluorescence pattern of antibodies from patients with double negative atypical IDD binding to astrocytes at rat cerebral cortex (green), similar to commercial GFAP‐IgG (red). (C) An immunofluorescence pattern of antibodies from patients with double negative atypical IDD binding to neurons at rat cerebral cortex (green), similar to commercial NeuN‐IgG (red). (D) An immunofluorescence pattern of antibodies from patients with double negative atypical IDD binding to oligodendrocytes at rat cerebellum (green), similar to commercial MOG‐IgG (red). (E) Seronegative immunofluorescence pattern. Nuclei were counterstained with 4′,6‐diamidino‐2‐phenylindole (DAPI) (blue). Scale bars: 100 μm. AQP4, aquaporin‐4; GFAP, glial fibrillary acidic protein; MOG, myelin oligodendrocyte glycoprotein; NeuN, neuronal nucleus.

    Article Snippet: Serum GFAP of double negative atypical IDD patients was quantified using commercial Human GFAP DuoSet ELISA (R&D Systems Minneapolis, MN, USA) by an independent investigator who was blinded to the result of TBA.

    Techniques: Immunofluorescence, Binding Assay